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MK-677's sustained receptor occupancy and non-peptide structure support different experimental designs from injectable peptide secretagogues.

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Indeed, since glutathione is involved not only in ROS and RNS scavenging but also cellular detoxification of additional reactive metabolic by-products such as methylglyoxal 34 , these results suggest that reactive metabolic by-products—which may include, but are not limited to, ROS and RNS—contribute to antibiotic-induced cell death, and that glutathione-mediated detoxification of these molecules contributes to protection from antibiotic lethality