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Additionally, gut wall injection of preformed, abnormal fibrils of αS led to the conversion of endogenous αS into the pathological phenotype 5 , resulting in the transfer of the pathologic αS into the central nervous system

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In contrast to the binding mode between c-MET and HGF, NK1 forms a stable head-to-tail dimer on its own, which recruits two c-METs to both sides in a symmetric manner (Fig