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Presented for research literature review only Circu ML et al
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First, targeting glycolysis can potentially increase mitochondrial metabolism and cell function, for example, by using the PDK inhibitor dichloroacetate [96] and PFKFB3 blocker 3-(3-pyridinyl)-1-(4-pyridinyl)-2-propen-1-one [97], however, this approach does not provide tissue-specific inhibition of glycolysis, which may cause off-target effects
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