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The new findings — identified using human plasma samples and mouse models of macular degeneration — suggest that increasing the amount of a molecule called apolipoprotein M (ApoM) in the blood fixes problems in cholesterol processing that lead to cellular damage in the eyes and other organs
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Even with anti-inflammatory and lipid-lowering interventions, the rupture of advanced atherosclerotic plaques remains a major risk factor for acute cardiovascular events and post-treatment recurrences (80)
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