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[DOI] [PubMed] [Google Scholar] 23.Stauch B., Johansson L.C., Cherezov V

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However, the in vitro data indicate that this cytotoxicity and the perforin expression levels in NK cells appear to be dependent upon endogenous IGF-1 and miR-483-3p expression, given that they can be inhibited by either IGF-1-neutralizing antibody or miR-483-3p overexpression

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