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As is likely for the initial events of the GRK- β -arrestin2 interaction, signaling by ERK1/2 may prevent coupling of MOR to effectors by phosphorylation of MOR at sites not occupied by G α -subunits (Schmidt et al., 2000)
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![PDF] Mercury elimination with oral DMPS, DMSA, vitamin C, and glutathione: an observational clinical review](https://figures.semanticscholar.org/c3561142922c93da4a18bcd6dbbd94f2069f2af2/2-Table1-1.png)
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