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Potent anti-fibrotic agent SalB suppressed autophagosome formation and autophagic flux in HSC by down-regulating the extracellular regulated protein kinases (ERK), c-Jun N-terminal kinase (JNK), and p38-MAPK pathways, leading to decreased expression of light chain 3β II, autophagy-related gene 5 (Atg5), α-SMA, and CoI ( SalB might be a useful and potent TGF-β antagonist to postpone the development of HF