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Other post-translational modification enzymes, such as RAS-converting enzyme 1 or isoprenyl carboxyl methyltransferase may also be potential therapeutic targets to block KRAS membrane localisation, especially phosphodiesterase-δ, which assists the transport of KRAS from the Golgi apparatus to the plasma membrane 155
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We further identified a putative ARE in the human CBS promoter region and verified that ARE is responsible for NRF2-activated CBS promoter-driven luciferase activity
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