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Additionally, this review underscores the importance of metabolic reprogramming, particularly hypoxia-driven adaptations and altered lipid metabolism, in fueling GBM progression and influencing the therapeutic response

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On the opposite, MCK1 deletion appeared to slightly enhance the methionine auxotrophy of CYS4 -OE cells (Figure 2C), suggesting that in the absence of Mck1p, Cys4p activity may be promoted