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However, excessive or dysregulated FoxO1 activation can lead to energy metabolism disorders and contribute to increased ketone body production through several mechanisms: first, by depleting oxaloacetate through its diversion to gluconeogenesis, FoxO1 activation reduces oxaloacetate availability for citric acid cycle function, thereby shunting excess acetyl-CoA toward ketogenesis
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[DOI] [PubMed] [Google Scholar] 58.Nzila A., Musa M.M., Sankara S., Al-Momani M., Xiang L., Li Q.X
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